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pylori infects more than 50% of the world's population (Taylor and Blaser, 1991). As one of the most common bacterial infection factors, H. pylori) was first identified in 1982 by Robin Warren and Barry Marshall, gastritis and peptic ulcer disease have been gradually approached as an infectious disease (Warren and Marshall, 1983 Suerbaum and Michetti, 2002). pylori intervention from emerging advances on ascorbic acid physiology and pharmacology. pylori infection, and outline some potential strategies for H. In this review, we summarize current understanding of the associations of vitamin C and H. Pharmacologic ascorbate can exert pro-oxidant effects locally as a drug by mediating hydrogen peroxide (H 2O 2) formation, which was applied to animal and clinical trials of cancer, sepsis, and severe burns etc. Recent studies suggest that ascorbate intake intravenously, but not orally pharmacologic ascorbate concentrations up to 30 mmol/L in blood, several millimolar in tissues as well as in interstitial fluid, are easily and safely achieved. pylori-eradication therapy are controversial. pylori eradication is shown to increase vitamin C levels, while the benefits of ascorbic acid oral intake to increase the effectiveness of H. Ascorbic acid supplementation likely relates to reduced incidences of bleeding from peptic ulcers and gastric cancer. pylori infected gastritis and peptic ulcers. Vitamin C levels both in gastric acid and serum have constantly been affirmed to be low in subjects with H. Numerous clinical reports show that ascorbic acid deficiency has been connect with gastritis. pylori), infection is predominantly known for its strong association with development of gastric diseases, including gastritis, peptic ulcers, and stomach cancer. The gram-negative bacterium, Helicobacter pylori ( H.